Are you in pain while walking, do you feel like your feet are cold, numb or itchy, do you have ulcerations that won’t heal or a family history of aorta aneurism (dilation)? The you need to evaluate your peripheral arterial system (namely those blood vessels that carry the blood to the legs), since there might be a connection between pain or numbness in legs, symptoms and disease.



In medical terms, the progressive narrowing of these vessels bears the name of obliterating peripheral arteriopathy of the lower limbs. It determines a decrease in functional capacity and affects the quality of life, while also leading to amputation of the legs and increasing the risk of death. Why? Because the main cause id arteriosclerosis and this is a (systemic) generalized process, where in most cases narrowing of the lower limb arteries coexists with lesions found in other territories (brain, kidneys, heart).



It is known at the moment that patients with pain or numbness in legs, symptoms and disease diagnosed as peripheral arterial disease have a 50% risk of suffering a heart attack, a 40% higher chance of suffering a stroke or renal failure, compared to the general population. Therefore, the more risk factors there are for arteriosclerosis the higher the risk is for you to develop this disease. Some of these risk factors can be corrected: smoking, arterial hypertension, diabetes, high cholesterol, while other risk factors cannot be corrected: age (patients aged over 50 develop this condition more often), sex (more men than women are patients) as well as heredity.

Rarely, other causes are noted, such as: an exaggerated vascular reactivity that determines arterial spasms, the occlusion of a vessel due to the migration of a blood clot or the existence of arterial dilation (aneurism) located most often in the aorta.

The most often symptom is the pain occurring while walking, which goes away, in the early stages, while pausing, but returns after walking approximately the same distance. This is called intermittent claudication. Its center varies depending on the narrowing of the vessels. Most often it is located in the calf, rarely in the thigh or buttocks, and in this latter case it can also be accompanied by sexual dysfunction. It will occur after smaller and smaller distances of walking until it starts appearing even in pauses, especially at night, at which point the patient tends to keep their leg hanging at the side of the bed.



Claudication, namely the pain caused by the narrowing of the arteries, needs to be distinguished from that produced by other diseases of veins, nerves, joints or muscles. Sometimes it is not typical, and can be felt as a tingling sensation, numbness or cold, which is why you should go to a doctor who, through simple maneuvers such as noticing the aspect of the teguments, feeling pulses, listening to arteries and determining arterial blood pressure in the arms and legs can tell you if you are suffering from arteriopathy or not.

Necrotizing fasciitis, also known as the flesh eating disease, represents a progressive infection, with necrotizing effect, of the fasciitis and of the subcutaneous tissue. This rare bacteria infection can destroy the skin and the underlying tissue, but it does not strike at the muscle. The infection progresses very fast and is usually accompanied by general toxic manifestations.



The most often incriminating etiological agent in the occurrence of flesh eating disease is Streptococcus Pyogenes (or group A strep). When the fasciitis is localized within the scrotum, perineum or genital region, it is called Fournier gangrene. The disease, although rare, is very serious: 30% of patients die if the infection is not treated adequately (Fournier gangrene comes with significantly higher mortality, some 75%). The average age of those who survive is 35, while patients aged above 50 fight off the infection with greater difficulty.



Flesh eating disease mostly targets adults (the average age of patients reaches 38-44), and children are only very rarely diagnosed with this disease. Most patients that develop this type of infection have a health considered to be satisfactory prior to the occurrence of the disease.

Signs and symptoms of flesh eating disease - The patient can develop intense pain after suffering a lesion of variable intensity and depth. Initially, the signs and symptoms of flesh eating disease decrease in intensity within 24 to 36 hours, but they intensely increase afterwards. Often, the pain felt is much more intense than it would be expected, considering the size of the initial wound. The patient could also claim fever, trembling, nausea and modifications in the abdominal transit (mostly manifested through the sudden occurrence of diarrhea).

Changes in the skin include redness in the area of the injury, painful tumefaction as well as increased temperature in the area targeted. If the infection is deep, the signs of inflammation occur later on. The symptoms evolve rapidly in seriousness and they occur suddenly, without premonition signs, as the infection spreads. This situation can become life threatening.

The develop complications including sepsis and shock (complications of the widespread tissue necrosis). Necrotizing fasciitis can even evolve towards multiple organ failure and death, if the symptoms are not recognized and the cause is not treated promptly and aggressively.

Diagnosis - Patients with necrotizing fasciitis show symptoms before going top the doctor. Clinical suspicion of necrotizing fasciitis occurs following careful analysis and the investigation of symptoms (especially if the rapid progression of the necrosis is recognized, as well as the worsening of the patient’s state in a relatively short amount of time).

For a certified diagnosis the doctor could collect a sample of necrotizing tissue. This tissue can be subject to microbiological analysis to precisely identify the bacteria responsible for the infection. In order to evaluate the health of the other organs and to establish the width and depth of the infectious process, the doctor will perform image testing (x ray scans, CR, MRI).



Therapeutic principles - Necrotizing fasciitis requires immediate and extremely aggressive treatment. Often, the condition requires supportive treatment to prevent the occurrence of liver, renal or respiratory failure, as well as shock. Medical treatment, based on antibiotics to which the bacteria is sensitive, is added with surgical treatment aimed at removing the dead tissue and stopping the expansion of the infection.

Preventive measures include washing the hands frequently, adequate hygiene and the rigorous cleaning of various injuries, scratches, burns or other types of lesions, penetrating or not.

Amyotrophic lateral sclerosis, also known as Lou Gehrig’s disease, is characterized through a progressive reduction of the number of nervous cells in the brain and spine, called motor neurons. The motor neurons regulate the contraction and relaxation of the voluntary muscles, which make the motion of the body possible.



ALS is a progressive disease, which is invalidating and fatal. Walking, speaking, eating, swallowing and other fundamental functions of the body become more difficult in time. These afflictions can cause various injuries and other complications.



One, maybe two, people in 100000 develop ALS every year. The men are affected slightly more often than women. Even though the condition can start at any age, it is more frequent in middle aged people and the elderly. The causes for ALS are unknown. Some 5-10% of people with ALS present an inherited form of the disease. ALS is not infectious.

Symptoms

Lou Gehrig’s disease early symptoms and later symptoms include: - weakness of lack of precision and dexterity in the hands and feet; - gradual loss of strength in the arms and legs; - inability of voluntary control of the arms and legs; - muscle spasms ; - rigid and unsteady walking; - difficulty with swallowing, swallowing and speaking; - fatigue; - muscle cramps, more frequent during the night, which can start later in the evolution of the disease; - pain in the final stages of the disease.

It is important to remember that Lou Gehrig’s disease early symptoms are not sure indicators of ALS, since these effects could be noted in many other circumstances as well.

The first sign of ALS is usually represented by a slight feeling of weakness in one leg, arm, the face or the tongue. Other problems are represented by greater imprecision and difficulty in the performance of actions that require precise movements of the fingers and hands. There can also be muscle spasms. The weakness extends to the arms and legs for months, or even years. While the neurons continue to decrease in numbers, the muscle cells that would have normally be stimulated by these nerves begin to decrease in number and the muscles atrophy.

Respiratory afflictions are perhaps some of the most frequent severe complications of ALS. While the muscles of the throat and chest atrophy, swallowing and coughing become more difficult, which sometimes triggers the aspiration of food or saliva into the trachea. This determines the occurrence of pneumonia (inflammation of the pulmonary tissue). Respiratory afflictions worsen as the disease progresses, increasing the risk of infections and respiratory insufficiency.



Pneumonia, pulmonary embolism, respiratory insufficiency and cardiac insufficiency (probably due to the inexistence of adequate respiration following the atrophy of the muscles) are the most common causes of death among ALS patients. In most cases, death occurs at the latest after three to six years from the initial symptoms, otherwise there are individual who survive with the disease even for decades. Even if there is no remedy for ALS, treatment may help maintain stability and independence, control of the symptoms and can avoid complications for as long as possible. The treatment is aimed at promoting emotional support as the invalidity increases.

The Alzheimer disease is a degenerative illness which affects certain areas of the brain that control memory, intelligence, and capacity of judgment, language and behavior. This disease represents the most common type of mental decline or dementia for elders.



The Alzheimer disease is a much serious affection than temporary memory loss which is characteristic to most elders. This disease is also associated with behavior disorders, personality disorders, loss of the ability to think correctly and loss of the capacity to perform daily tasks.



Family and friends notice in the beginning these changes, but these changes can be noticed by the patient in cause. The cause of Alzheimer disease remains unknown, but there could be several factors which could lead to this disease. Some of the deteriorations which occur in some areas of the brain are connected to the loss of chemical messengers of the neurons. The cause of these modifications is unknown, but scientists are still researching in order to determine these causes. Even though most of the patients do not have this disease in their family medical background, the risk of developing the Alzheimer disease is increased at patients who have a relative suffering of this disease.

Some studies suggested the fact that zinc and aluminum might have a role in starting this disease, but these theories have not been supported by proof. Memory loss is most of the times one of the first symptoms of Alzheimer’s disease. Many elders are worried when they have short term memory loss. Some of episodes occur at people older than 60- 70 years old, but only a small part of them can develop the Alzheimer disease. If you notice you or someone close having short memory loss episode you should consult a specialist doctor.

Normal short term memory loss episodes include: forgetting a certain experience, forgetting where you parked your car, forgetting events from your past, the name of a person which you will recall later or forgetting where you left an object, like your car keys. Some of the early symptoms of Alzheimer’s disease are: forgetting you ever had an experience, forgetting how to read the clock or drive your car, forgetting about current experiences or the fact that you left the gas on or even forgetting you know a person.

There is little evidence of the fact that maintaining a special intellectual activity might diminish the risk of developing the Alzheimer disease. Some activities like reading the newspaper, books, magazines, playing society games, solving puzzles, visits to the museum or even listening to the radio can help in preventing the development of Alzheimer disease.



The more scientists are uncovering the causes of Alzheimer’s disease, the more they will be able to find a cure as well as a way to prevent it. The medication used for preventing some disease will be available for preventing the Alzheimer’s disease or for people who have an increased risk of developing this disease. The scientists are trying now to develop a vaccine for Alzheimer.

Hodgkin’s disease is a type of malignant lymphoma that especially attacks the ganglions of the throat and armpit. It often occurs in young adults and elderly people. As opposed to several years ago, today’s Hodgkin’s disease has a pretty high cure rate. So if you are suffering from this disease or have just been diagnosed with it, then you should not despair, because there are solutions for you out there.



Here I will tell you about the nature of this disease, its origins, the Hodgkin ’s disease progressive symptoms, diagnosis, treatment and evolution.



What is this disease? Hodgkin’s disease is a form of lymphoma, therefore a disease that affects the lymph nodes. It usually develops in young people with ages between 15 and 34, or in elderly people aged over 60. The occurrence of the disease is higher in Europe and in the United States of America than it is in Asia, and also, the incidence is higher in well developed countries as opposed to countries that are now developing.

Origins of the disease - It is not exactly known what is the element that triggers this disease or where it comes from. It seems that the disease is not contagious. Specialists often assume that the favorable grounds for the development of this disease are secured by infection with the Epstein-Barr virus.

Hodgkin’s disease progressive symptoms - The main symptom is the swelling of the ganglions. This swelling is not painful and does not present an apparent infection. These ganglions are especially situated in the cervical area and in the armpit. The increase in volume of the ganglions is sometimes, but not always, accompanied by symptoms such as:
- fever, which can persist for several days, then disappear for weeks in a row, and then return suddenly and with no apparent cause; - sweating during the night; - loss of weight; - high levels of physical fatigue; - depending on the location of the ganglions afflicted, there can be other symptoms. For example, if the ganglions are placed in the chest, on a thorax level, there can coughing or chest pains.

Diagnosis - Aside from the clinical exam, a biopsy is the one that allows a diagnosis, added with blood tests performed by the doctor.

Treatments that are currently available - The treatment of Hodgkin’s disease involves radiotherapy or chemotherapy, and the two are often associated, or combined, in the treatment scheme.



The evolution of Hodgkin’s disease - Today, Hodgkin’s disease is well treated in the wide majority of cases. There are four stages to this illness, from I, which is the least advanced, to IV, which is the most severe. For stages I and II, the cure rate is higher than 80%. The cure rate reaches between 70% and 80% in stage III and a percentage of 50% in stage IV, a level where one in two cases I cured. In exchange, the chemotherapy used in the treatment of Hodgkin’s disease can sometimes have late consequences, sometimes triggering leukemia, lung cancer or stomach cancer.

The Parkinson disease can affect one’s capacity of controlling the movements of their body. Some nervous cells inside the brain normally produce a chemical substance called dopamine. Dopamine is a chemic mediator or neurotransmitter through which the nervous cells control the movement of the body.



In Parkinson disease, these nervous cells which produce dopamine, self destruct and lead to low amounts of dopamine. When this starts to happen, the body can start showing sign of difficulty in even the simplest movements. The Parkinson disease comes with a wide range of symptoms and complications.



Treatment of the Parkinson disease can control the symptoms in the early stages of the disease and it is applied as soon as the symptoms are affecting the daily activities and an ill person’s ability to work. For example, a right handed person who starts trembling on the left side might not feel bothered by this symptom and they might not feel the need for treatment until the symptoms start to worsen and start growing in intensity.

A person who is not able to fulfill their daily tasks because of the symptoms of Parkinson disease will want to start treatment faster than a person who does not work or who is not affected in their daily tasks by the symptoms of Parkinson disease. The symptoms of Parkinson disease are usually found on people aged 50 to 60 year old. The affection evolves slowly and sometimes can not be noticed by family, friends or even the person who has the Parkinson disease. A small number of people develop the Parkinson disease symptoms on one side of the body and most of them usually do not cross to the other side of the body.

Medication can somewhat control the symptoms of Parkinson disease, but it becomes more and more inefficient as the disease evolves. The Parkinson disease can cause a wide range of complications as the disease evolves. The connection between the Parkinson disease and the risk factors is still in question. Some of these factors can be: genetic factors, age, toxins from the environment or antioxidants. Even if research is about to reveal some answers, the experts do not know for sure what is it that causes the Parkinson disease.

There are studies, which are still in development, and that are trying to identify a cause for the Parkinson disease. Only a small percentage of the people diagnosed with the Parkinson disease have a parent, brother or sister who have this disease. However, the transmission of some abnormal genes seems to be the cause of the Parkinson disease which starts precocious in some families on which this affection is discovered at younger ages.



The low levels of dopamine- the chemic mediator- involved in the control of movements lead to the symptoms of Parkinson disease. The lack of this neurotransmitter shows up when the nervous cells from one side of the brain- the black substance- which produce dopamine start to self destruct or deteriorate. The exact cause of this affection of the nervous cells is unknown to specialist and researchers.

In general terms, the septum is a wall that separates two cavities. In the nose, the septum is a thin wall which separates the right nostril from the left nostril and is meant to support the nose and direct the flow of air.



In the upper segment, the nasal septum is formed of a thin bone, and in the lower segment by a cartilage. Normally, this structure is relatively straight and positioned at the center of the nose. When the nasal septum is sufficiently shifted to one of the sides, there can be a breach the flow of air and the amount of mucus in the nose. This condition, called a deviated septum, can determine what is called septum symptoms of disease.



Septum symptoms of disease - A deviated septum can simply be a variation of the normal structure and can occur during prenatal development, during child birth or as the nose grows. Also, the affliction can occur as result of nasal trauma. In most cases, deviated septum does not produce any symptoms and does not require treatment. Still, when the shift is severe, it can obdurate the passage of air through the nostrils and predispose to repeated infections in the sinuses, bleeding, snoring or apnea during sleep.

Diagnosis: it is relatively easy to spot a deviated septum, but it is much more difficult to establish if the deviation requires correction. Usually the patient complains about not being able to breathe except through one nostril.

Medicated treatment - Treatment depends on the symptoms claimed by the patient: the doctor can use pain killers to ease the headache, nasal decongestants to reduce secretions, antibiotics against infection or, if the symptoms are persistent, corrective surgery for the reconstruction of the nasal septum.

Surgical intervention - Septoplasty is the surgical intervention performed with the purpose of correcting a deformed nasal septum and thus removing the obstruction of nasal passages. The operation can be performed under general or local anesthetics, based on the preference of the patient and the indications of the doctor. The operation takes some 60 to 90 minutes and, usually, the patient leaves the hospital after 3 or 4 hours. The surgeon will perform an incision within the nose and will remove the mucous membrane whi covers the cartilage and the bone. The deformed septum will be cut out or rectified, after which the mucous membrane is placed back on the septum.



Since septoplasty is usually performed under local anesthetics, there is no need for elaborate preparations; still, you should be prepared for some blood tests, radiological investigations and a detailed examination of the nose. If you are hypertensive, have diabetes or afflictions which predispose to excessive bleeding, there will be special precaution taken before the surgical intervention. Ten days before the operation you are recommended to cease the administration of medicine that could increase the risk of bleeding, such as aspirin or other non-steroid anti-inflammatory. After the intervention you will feel slight sensitivity or pain in the nose. The doctor will recommend the drugs needed to ease the pain.

Ischemia of the intestine is the final result of interruption or reduction of blood perfusion in the intestine in question. Still, the clinical manifestations of intestinal ischemia vary from light chronic symptoms   to acute dramatic episodes, depending on the involved segment, the expansion of the occlusion or ischemia and the speed of development for the process.

The clinician needs to have knowledge on the spectrum of clinical manifestations. The intestine receives its arterial vascularization from branches in the celiac network and from the upper and lower mesenteric arteries. So there is a connection between ischemia of the intestine and mesenteric artery disease symptoms. The small intestine is irrigated by the celiac and upper mesenteric arteries. A wide network of vessels as well as the possible development of collateral circulation determine the clinical panel of acute or chronic intestinal arterial insufficiency.

Acute intestinal ischemia can be classified as occlusive or non-occlusive. The occlusion can result through thrombosis or arterial embolism of the mesenteric or celiac arteries, or through venous occlusion with the same distribution. Arterial embolism occurs mainly in patients with chronic or recurrent arterial fibrillation, valve prosthetics or illnesses of the valve. Arterial thrombosis is usually associated with excessive arteriosclerosis or with a low hart rate.

Venal occlusion is rare; it is occasionally encountered in women who take oral contraceptives. Some half of patients with mesenteric ischemia do not have definite occlusion of a main vessel, a situation called non-occlusive ischemia. The exact cause of this affliction is obscure; systemic arterial hypotension, cardiac arrhythmias, prolonged cardiac insufficiency, digitalic treatment and dehydration can constitute some of the main factors involved in the development of the condition.

Mesenteric artery disease symptoms include severe abdominal pain, often colicative and periombilical in early stages, later becoming diffuse and constant. Throwing up, anorexia, diarrhea and constipation are also frequent symptoms when faced with this disease, but their diagnostic value is pretty low, since they are often encountered in other types of afflictions as well. An examination of the abdomen can indicate sensitivity and distension. Intestinal noises are often normal, even in the presence of severe heart attack.

Some patients have, during clinical examination, a surprisingly normal abdomen in contrast with the severe abdominal pain they are claiming. Light bleeding in the gastric intestinal area is often detected through the examination of the fecal matter, looking for occult bleeding. Massive hemorrhaging is unusual, except for the cases where the patient is suffering from ischemic colitis.

One typical laboratory element Is marked leucocytosis. Later in the evolution of the disease, after 24 to 72 hours, there is gangrene in the intestine, with diffuse peritonitis, sepsis and shock. A simple abdominal radiography in patients with mesenteric ischemia can point out distension. The radiological examination of the intestine using barium shows non-specific dilation, reduced motility and the presence of thick mucous cleavages, similar to finger prints. Acute mesenteric ischemia is a severe clinical affliction with high morbidity and mortality rates. Patients suspected of acute arterial embolism need to be examined immediately through a celiac and mesenteric angiography to locate the embolus, followed by an embolectomy.

Diseases of the urinary apparatus in dogs include: congenital anomalies; infectious diseases; intestinal nephritis; non-infectious diseases; renal blockage; metabolic illnesses; renal neoplasia; urolithiasis, etc.



Factors that can influence the appearance of renal diseases in dogs include: -age – the chances for renal blockage to occur grow with age in dogs, and they double once the animals reach the age of 10 - 15; -race – renal afflictions are more common in certain races of dogs, which can predispose the animal to the occurrence of renal dysfunction. These races include: Cocker Spaniel, Lhasa Apso, Samoyede and Doberman Pinscher.;



-the environment – certain common chemical in the environment (phenol based disinfectants, thawing agents, led based paint) and also certain types of medicine are toxic for the kidneys; -the diet – a careful examination of the amounts of phosphorus and protein in the food can slow the progression of renal illnesses in dogs who have already been diagnosed with renal problems.

Stages of renal disease: -normal renal function – when some of the nephrons are lost but their function is covered by the remaining nephrons, and there are no clinical signs of disease; -renal insufficiency (primary stage of disease) – the animal is not able to sufficiently concentrate the urine and thirst is higher than usual; -early renal blockage – toxic metabolites (residual substances) are accumulated in the body because the kidneys cannot eliminate them in an efficient manner, thus determining the occurrence of some of the earliest symptoms of kidney disease in dogs; -advanced renal blockage –there are obvious signs of disease, reaching all the way to collapse and death.

Common diseases of the uro-genital apparatus and earliest symptoms of kidney disease in dogs:

Urolithiasis - Urolithiasis or urinary calculosis is a common illness in dogs. Causes: in dogs, urine is a hypertonic solution, due to the various crystalloids, the uric acid for example, which is difficult to dissolve. Signs of the disease: there is a clear differentiation between the symptoms depending on the location of the calculi. Thus, still renal calculi do not produce any symptom of disease. The rugous and mobile calculi can damage the renal basin, producing various infections; calculi located on urethras can produce colic (dramatic abdominal pain); calculi in the urinary bladder can produce hemorrhagic cystitis.

Calculi located in the urethra can partially or completely obdurate the urethra, causing the dog to lose urine, in the first case, or not to urinate at all, in the second case. Urethral calculi in the male dog, which stop at the base of the penian bone are extracted by the doctor, in the veterinarian clinic, through a surgical procedure.



Urolithiasis and bladder calculi - Several factors contribute to the formation of sand and stones in the urinary apparatus. The growth in salt concentration in the urine determines the depositing of the calculi, layer upon layer, on a renal level or in the urinary bladder. Normally, the urine of dogs and cats is slightly acid. On the other hand, most salts are poorly soluble in alkaline solutions, which is why, the more alkaline the urine becomes, the more will the animal in question be prone to developing some form of sand or uroliths. A series of bacteria decompose the urea from the urine into ammonia, a compound that renders the urine alkaline.

Celiac disease is an affection of the immune system, triggered by the ingestion of gluten in aliments. Gluten leads to lesions of the intestine. Gluten is a vegetal protein which can be found in cereals like rice, wheat, barley or rye. The lesions of the intestine can affect the normal absorption of nutrients, especially fats, calcium and iron.



Celiac disease is also known as Sprue celiac. The cause that triggers the Celiac disease is unknown, but medical search in the last years identified some genes which facilitate the risk of Celiac disease. People who have a mother, father, sister, brother, son or daughter with Celiac disease have a greater risk of developing this disease. External factors, like viral or bacterial infections, can cause several modifications of the intestine, especially in the case of those who are genetically predisposed to Celiac disease. Eating aliments which contain gluten triggers several immune reactions which can cause the intestine lesions that are characteristic for this disease. These lesions determine digestive disorders and malnutrition.



Actual genetic studies are focused on the connection between genes, immune and external factors. Research performed until now were not able to prove the real age of this disease or the mechanism of complications characteristic to Celiac disease. The symptoms of Celiac disease differ from case to case, from easy symptoms which are barely noticeable, to severe symptoms and complication which have a negative impact over daily life.

The symptoms of Celiac disease are: stomach bloating, meteorism, stomach discomfort- caused by bad digestion of the intestine, gastric pain, abnormal stool, diarrhea (the stool has a shinny and bright aspect and smells bad), severe weight loss, in spite of a good appetite, fatigue and weakness and throwing up.

Celiac disease can also lead to osteoporosis and other bone disease, infertility and lack of menstruation, delayed puberty, frequent respiratory infections, memory and concentration difficulty, mental disorders, like irritability in children and depression in adults. All these symptoms, which are unspecific, can most of the time delay the proper diagnosis.

The risk of Celiac disease is increased for people who have antecedents in their family. It has been proven scientifically, that people who have a first degree relative with Celiac disease, like mother, father, sister, brother, children, can developed the Celiac disease as well. People with celiac disease usually have other affections as well, like: dermatitis- frequent in adults, but can be found in children as well, osteoporosis, type I diabetes, auto immune thyroid, Down syndrome, Sjogren syndrome, antibody deficit or anemia.



There are lots of people interested in how to ease celiac disease symptoms. As a doctor, I have been asked several times how to ease celiac disease symptoms, and here is what I can tell you. Most of the times, a medication treatment is not required for celiac disease. In the case of children, a diet of iron and calcium is required and can ease the symptoms of celiac disease. Once you remove gluten from their diet, the symptoms usually disappear in 2 or 3 weeks. Once the intestine recovers, you can consider yourself cured. Medication is required only if there are severe complications and the symptoms persist.